Category Archives: Psoriasis

Psoriasis Treatments Revitol Dermaisis Review and Bonus Package Launched
Psoriasis Treatments: Revitol Dermaisis Review and Bonus Package Launched December 10 2014 http://productreviewsreport.com/psoriasistreatments ...

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How To Permanently Eliminate Psoriasis | Amazing Guide About How To Permanently Eliminate Psoriasis
http://www.tinyurl.com/psoriasisfreeforlifenow How To Permanently Eliminate Psoriasis: Psoriasis Free for Life is an effective method written by Katy Wilson, who is an alternative Medical Practitio...

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About Psoriasis _ What is Psoriasis How To Cure Psoriasis.mp4
About Psoriasis _ What is Psoriasis How To Cure Psoriasis.mp4.

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Download Living with Psoriasis PDF
Click To Download PDF Here : http://bit.ly/1D1YCD8.

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At mypsoriasis.co.nz youll find suggestions for everything from how to handle kids awkward questions to dealing with an over-protective family.

Often, the stress of keeping feelings bottled up can make psoriasis symptoms worse, so the more help you can get the better youll be able to manage it. Remember, youre not alone. Theres a whole psoriasis community there to support you.

The information youll find at mypsoriasis.co.nz recognises symptoms can affect many decisions and aspects of your life and that it can take an emotional toll because of its long-term and visual nature. Youll be able to download the booklet Refusing to hide the insiders guide to psoriasis which is full of useful information to help you better understand and manage your psoriasis.

Youll also find loads of expert information from dermatologists, nurses and psychologists, along with personal stories of people just like you who share their experience of and strategies for living with the psoriasis.

As you probably already know, psoriasis is an autoimmune disease. The bodys immune system is mistakenly reacting to some of your own bodys cells, resulting in accelerated skin cell growth.

Normal skin cells replace themselves every 28 to 30 days, but skin cells affected by psoriasis mature in just 3 to 4 days and pile up instead of falling away on their own. The resulting build-up forms the silvery, scaly, red patches called plaque psoriasis.

While genetics play a role in psoriasis, scientists believe exposure to environmental triggers may propel the disease into action. For example, you may experience a worsening of your symptoms in the winter and improvement in the summer. Other triggers may include injury, stress, certain medicines and some types of infections.

Your best tool is information, plenty of which can be found at mypsoriasis.co.nz.

For example, did you know psoriasis affects more than just skin? Around 30 percent of people with psoriasis develop psoriatic arthritis that can also affect the joints, causing them to become stiff, painful and swollen. More information about psoriasis can be found mypsoriasis.co.nz.

As for how to handle those curious kids questions? How about: I have extra-happy skin. My skin cells are so happy they jump off after only a few days! Brilliant!

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Support for psoriasis community in new website

Psoriasis Free For Life Program | Amazing Psoriasis Free For Life Program By Katy Wilson
http://www.tinyurl.com/psoriasisfreeforlifenow Psoriasis Free For Life Program: Psoriasis Free for Life is an effective method written by Katy Wilson, who is an alternative Medical Practitioner...

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Psoriasis Revolution and Discount Link
Discount Link http://www.psoriasisrevolutionpdf.com/-Psoriasis-Revolution- About the Book Psoriasis Revolution is a well-written book that carefully describe...

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It is estimated that about 35 million people worldwide currently suffer from dementia and it is expected that the number will increase to 135 million by the year 2050. The disease is already one of the most common health problems in the elderly, which is why experts predict that the numbers of people affected will increase over time. Researchers at the Department of Psychiatry and Psychotherapy of the University Medical Center of Johannes Gutenberg University Mainz (JGU) have recently gained new insights into how it may in future be possible to treat patients with the currently most common form of dementia, Alzheimer's disease. It seems that a drug that is actually approved for treatment of the dermal disorder psoriasis stimulates the activity of the enzyme ADAM10 in the brain of Alzheimer's patients. There is already good evidence from basic research that this enzyme should be capable of suppressing Alzheimer's disease-related effects such as impaired cerebral function and that it thus might improve learning and memory capacity in patients. The results of the related study have recently been published in the journal Neurology.

According to estimates of the German Alzheimer's Association (DAlzG), approximately 1.5 million dementia patients currently live in Germany. Some 1 to 1.2 million of these suffer from Alzheimer's. Medicine is currently only able to treat the symptoms of the disease and delay its progress and thus also the need for increased nursing care. No curative therapy has yet been developed. This means that Alzheimer's disease remains one of the biggest challenges to modern medicine and is an important field for research.

There is still no consensus on what triggers the most common form of the disease, late-onset Alzheimer's. However, it is generally accepted that the activity of certain enzymes called secretases plays a role here. These enzymes cleave proteins on cell membranes, releasing the products of this cleavage process into the extracellular space. What happens in Alzheimer's is that there is increased cleavage of the amyloid precursor protein by beta-secretase, leading to the formation of amyloid-beta peptides. These peptides aggregate, damage nerve cells, and are the main component of the so-called Alzheimer's plaques that accumulate in the brains of patients. The alpha-secretase ADAM10 is a competitor of beta-secretase. It cleaves the amyloid precursor protein in such a way that the synthesis of amyloid beta-peptides is prevented while the growth factor APPs-alpha, which protects nerve cells, is released.

Taking this information as their starting point, Dr. Kristina Endres and Professor Falk Fahrenholz of the Department of Psychiatry and Psychotherapy of the Mainz University Medical Center have decided to take a new approach to the treatment of Alzheimer's. Working in collaboration with Professor Klaus Lieb and Professor Andreas Fellgiebel, both also working at the Department of Psychiatry and Psychotherapy, and with the cooperation of Professor Stefan Teipel and his team at the German Center for Neurodegenerative Diseases (DZNE) in Rostock, the researchers have demonstrated that oral administration of a psoriasis medication in a group of Alzheimer's patients results in elevated levels of APPs-alpha in their spinal fluid. This is interpreted as a stimulation of the activity of the alpha-secretase ADAM10, which in turn would result in the reduced accumulation of Alzheimer's plaques. In animal models of Alzheimer's disease, it has also been shown that ADAM10 enhances learning and memory capacity. The medication was well-tolerated by the patients. In order to further investigate the effect of the test substance on cognitive performance and to establish whether it can be used as a long-term treatment for Alzheimer's patients, larger clinical trials in which the substance is administered for longer periods will need to be undertaken.

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The above story is based on materials provided by Johannes Gutenberg Universitaet Mainz. Note: Materials may be edited for content and length.

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New approach for treating Alzheimer's disease: Psoriasis drug

December 8, 2014

Provided by Dr. Kristina Endres, Mainz University Medical Center

It is estimated that about 35 million people worldwide currently suffer from dementia and it is expected that the number will increase to 135 million by the year 2050. The disease is already one of the most common health problems in the elderly, which is why experts predict that the numbers of people affected will increase over time. Researchers at the Department of Psychiatry and Psychotherapy of the University Medical Center of Johannes Gutenberg University Mainz (JGU) have recently gained new insights into how it may in future be possible to treat patients with the currently most common form of dementia, Alzheimers disease. It seems that a drug that is actually approved for treatment of the dermal disorder psoriasis stimulates the activity of the enzyme ADAM10 in the brain of Alzheimers patients. There is already good evidence from basic research that this enzyme should be capable of suppressing Alzheimers disease-related effects such as impaired cerebral function and that it thus might improve learning and memory capacity in patients. The results of the related study have recently been published in the journalNeurology.

According to estimates of the German Alzheimers Association (DAlzG), approximately 1.5 million dementia patients currently live in Germany. Some 1 to 1.2 million of these suffer from Alzheimers. Medicine is currently only able to treat the symptoms of the disease and delay its progress and thus also the need for increased nursing care. No curative therapy has yet been developed. This means that Alzheimers disease remains one of the biggest challenges to modern medicine and is an important field for research.

There is still no consensus on what triggers the most common form of the disease, late-onset Alzheimers. However, it is generally accepted that the activity of certain enzymes called secretases plays a role here. These enzymes cleave proteins on cell membranes, releasing the products of this cleavage process into the extracellular space. What happens in Alzheimers is that there is increased cleavage of the amyloid precursor protein by beta-secretase, leading to the formation of amyloid-beta peptides. These peptides aggregate, damage nerve cells, and are the main component of the so-called Alzheimers plaques that accumulate in the brains of patients. The alpha-secretase ADAM10 is a competitor of beta-secretase. It cleaves the amyloid precursor protein in such a way that the synthesis of amyloid beta-peptides is prevented while the growth factor APPs-alpha, which protects nerve cells, is released.

Taking this information as their starting point, Dr. Kristina Endres and Professor Falk Fahrenholz of the Department of Psychiatry and Psychotherapy of the Mainz University Medical Center have decided to take a new approach to the treatment of Alzheimers. Working in collaboration with Professor Klaus Lieb and Professor Andreas Fellgiebel, both also working at the Department of Psychiatry and Psychotherapy, and with the cooperation of Professor Stefan Teipel and his team at the German Center for Neurodegenerative Diseases (DZNE) in Rostock, the researchers have demonstrated that oral administration of a psoriasis medication in a group of Alzheimers patients results in elevated levels of APPs-alpha in their spinal fluid. This is interpreted as a stimulation of the activity of the alpha-secretase ADAM10, which in turn would result in the reduced accumulation of Alzheimers plaques. In animal models of Alzheimers disease, it has also been shown that ADAM10 enhances learning and memory capacity. The medication was well-tolerated by the patients. In order to further investigate the effect of the test substance on cognitive performance and to establish whether it can be used as a long-term treatment for Alzheimers patients, larger clinical trials in which the substance is administered for longer periods will need to be undertaken.

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Source: Provided by Dr. Kristina Endres, Mainz University Medical Center

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Psoriasis Medication May Be Effective in the Treatment of Alzheimer's

08.12.2014 - (idw) Johannes Gutenberg-Universitt Mainz

Psoriasis medication may be effective in the treatment of Alzheimer's disease It is estimated that about 35 million people worldwide currently suffer from dementia and it is expected that the number will increase to 135 million by the year 2050. The disease is already one of the most common health problems in the elderly, which is why experts predict that the numbers of people affected will increase over time. Researchers at the Department of Psychiatry and Psychotherapy of the University Medical Center of Johannes Gutenberg University Mainz (JGU) in Germany have recently gained new insights into how it may in future be possible to treat patients with the currently most common form of dementia, Alzheimer's disease. It seems that a drug that is actually approved for treatment of the dermal disorder psoriasis stimulates the activity of the enzyme ADAM10 in the brain of Alzheimer's patients. There is already good evidence from basic research that this enzyme should be capable of suppressing Alzheimer's disease-related effects such as impaired cerebral function and that it thus might improve learning and memory capacity in patients. The results of the related study have recently been published in the journal Neurology.

According to estimates of the German Alzheimers Association (DAlzG), approximately 1.5 million dementia patients currently live in Germany. Some 1 to 1.2 million of these suffer from Alzheimer's. Medicine is currently only able to treat the symptoms of the disease and delay its progress and thus also the need for increased nursing care. No curative therapy has yet been developed. This means that Alzheimer's disease remains one of the biggest challenges to modern medicine and is an important field for research.

There is still no consensus on what triggers the most common form of the disease, late-onset Alzheimer's. However, it is generally accepted that the activity of certain enzymes called secretases plays a role here. These enzymes cleave proteins on cell membranes, releasing the products of this cleavage process into the extracellular space. What happens in Alzheimer's is that there is increased cleavage of the amyloid precursor protein by beta-secretase, leading to the formation of amyloid-beta peptides. These peptides aggregate, damage nerve cells, and are the main component of the so-called Alzheimers plaques that accumulate in the brains of patients. The alpha-secretase ADAM10 is a competitor of beta-secretase. It cleaves the amyloid precursor protein in such a way that the synthesis of amyloid beta-peptides is prevented while the growth factor APPs-alpha, which protects nerve cells, is released.

Contact: Dr. Kristina Endres Department of Psychiatry and Psychotherapy Mainz University Medical Center Langenbeckstr. 1 D 55131 Mainz, GERMANY phone +49 6131 17-2133

Press contact Barbara Reinke Press and Public Relations Mainz University Medical Center Langenbeckstr. 1 D 55131 Mainz, GERMANY phone +49 6131 17-7428 fax +49 6131 17-3496 e-mail: pr@unimedizin-mainz.de Weitere Informationen:http://www.uni-mainz.de/presse/17845_ENG_HTML.php - press release

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Researchers at Mainz University explore new approach for treating Alzheimers disease