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Category Archives: Transhuman News
Stubborn Psoriasis vs TWEEZERS! – Video
Posted: April 11, 2015 at 7:44 am
Stubborn Psoriasis vs TWEEZERS!
After lubing up with some coconut oil in my last video, I found this patch on my left ankle.
By: Huy Ngo
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Stubborn Psoriasis vs TWEEZERS! - Video
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Testimonios jaquecas y psoriasis – Video
Posted: at 7:44 am
Testimonios jaquecas y psoriasis
By: Nutricin Inteligente
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Testimonios jaquecas y psoriasis - Video
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How to GET RID of PSORIASIS with a PLANT BASED VEGAN DIET – Video
Posted: at 7:44 am
How to GET RID of PSORIASIS with a PLANT BASED VEGAN DIET
In this video I explain how you can get rid of psoriasis naturally using a plant based vegan diet and alkalising regime. Things to remove or eliminate: Refined sugar Dairy Meat Eggs Nightshade...
By: Jon Maddison
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How to GET RID of PSORIASIS with a PLANT BASED VEGAN DIET - Video
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Psoriasis Revolution Treatment Does It Work – Video
Posted: at 7:44 am
Psoriasis Revolution Treatment Does It Work
Psoriasis Revolution by Dan Crawford download online: http://tinyurl.com/PsoriasisRevolutionDownloadPDF Restore your natural inner balance and prevent untreated Psoriasis-related health issues...
By: Gillevet Dilan
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Download Handbook of Psoriasis PDF – Video
Posted: at 7:44 am
Download Handbook of Psoriasis PDF
Download Link : http://bit.ly/1D9A8pL Login to the website and feel free to download the PDF. Please register first if you don #39;t have an account.
By: Gilda Sophia
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Download Handbook of Psoriasis PDF - Video
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Epsom Salt Detox Bath Recipe: For Weight Loss, Sore Muscles, Psoriasis, Eczema & Flu 1 – Video
Posted: at 7:44 am
Epsom Salt Detox Bath Recipe: For Weight Loss, Sore Muscles, Psoriasis, Eczema Flu 1
WANT TO KNOW MORE? = http://tinyurl.com/lu5s33k Epsom salt detox bath recipe benefits for weight loss, sore muscles, psoriasis, eczema and flu. Epsom salt bath detoxes the body very effectively...
By: Jeanette Lalouree
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Epsom Salt Detox Bath Recipe: For Weight Loss, Sore Muscles, Psoriasis, Eczema & Flu 1 - Video
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Coffee Chat: 4-9-15 Q&A; Psoriasis, Spring Preps – Video
Posted: at 7:44 am
Coffee Chat: 4-9-15 Q A; Psoriasis, Spring Preps
Hey guys, just a quick coffee chat. Thanks for watching, and God bless. J.
By: jnull0
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Coffee Chat: 4-9-15 Q&A; Psoriasis, Spring Preps - Video
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CME Outfitters Two-Part Medical Simulation on Psoriasis Featuring Expert Faculty Commentary Now Available Online
Posted: at 7:44 am
Bethesda, Maryland (PRWEB) April 09, 2015
CME Outfitters (CMEO), a leading accredited provider in continuing medical education, announces its newest CMEO Medical Simulations, Not Just Skin Deep: Psoriasis Parts I and II. Faculty experts April W. Armstrong, MD, MPH, and Alan Menter, MD guide you through the simulations to improve your knowledge base and clinical management skills in patients with psoriasis, and improve your knowledge of psoriasis comorbidities and clinical strategies to improve long-term patient outcomes. Dermatologists, physicians, and other health care professionals, including pharmacists, nurses, physician assistants, and nurse practitioners who have an interest in psoriasis are encouraged to participate.
Patients with moderate-to-severe psoriasis are at risk for a variety of comorbidities. Dermatologists are in a unique position to screen for these comorbidities and counsel patients to be aware of early symptoms and risk factors. Prevention and early intervention can be achieved by appropriate treatment of psoriasis and reduction of other risk factors contributing to these complications. In this two-part medical simulation, you will meet Lisa, a 45-year-old woman who presents to you feeling very self-conscious about her skin condition, in part one, and has questions about the effects of her skin condition on her overall health, in part two. Navigate through the simulation and Lisas clinical challenges with Dr. Armstrong and Dr. Menter as your guides.
At the end of this activity, participants should be able to:
Click here for more information about the faculty, financial support, credit information, disclosures, and to participate today!
About CME Outfitters, LLC CME Outfitters develops and distributes live, recorded and web-based, outcomes- and evidence-based educational activities to thousands of clinicians each year and offers expert accreditation and outcome services for non-accredited organizations. CME Outfitters focuses on delivering education to specialty audiences, with strong expertise in neuroscience, inflammatory, infectious, and autoimmune diseases, and cardiovascular disease. For a complete list of certified activities and more information, visit http://www.cmeoutfitters.com or call 877.CME.PROS (877.263.7767).
CME Outfitters Improving Clinical Behavior One Change at a Time
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Ingredient in MS, Psoriasis Drugs Linked to Two Deadly Brain Infections
Posted: at 7:44 am
By Dennis Thompson HealthDay Reporter Latest Infectious Disease News
WEDNESDAY, April 8, 2015 (HealthDay News) -- An active ingredient in some psoriasis and multiple sclerosis medications has been linked to two cases of a rare and sometimes lethal brain infection.
The ingredient, dimethyl fumarate, appears to have contributed to the deaths of two European women. The women contracted progressive multifocal leukoencephalopathy, or PML, according to two letters published in the April 9 issue of the New England Journal of Medicine.
One case involved a 54-year-old woman with multiple sclerosis. She died in October 2014 from complications related to PML and pneumonia, following 4.5 years of treatment with a time-delayed form of dimethyl fumarate carrying the brand name Tecfidera, researchers reported.
The second case was a 64-year-old woman with psoriasis. She died in August 2014 from PML after being treated with a delayed-release dimethyl fumarate compound with the brand name Psorinovo for two years, according to the researchers.
These cases follow reports linking other drugs containing dimethyl fumarate with PML, including Tysabri and Fumaderm, the researchers said.
However, the latest case reports do not prove that dimethyl fumerate caused the PML infections.
And outside experts said cases of PML occur rarely enough that dimethyl fumarate should remain on the market, as an option for people with MS or psoriasis.
"It's something to be concerned about and something to pay attention to. But from what we know right now, the occurrence of PML appears to be pretty low for Tecfidera," said Bruce Bebo, executive vice president of research for the National Multiple Sclerosis Society.
PML is caused by the JC virus, which normally lies dormant in most people's bodies and causes no harm. But if a person's immune system becomes compromised, the JC virus can flare up and attack the white matter of the brain.
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Stem cell disease model clarifies bone cancer trigger
Posted: at 7:44 am
Using induced pluripotent stem cells (iPSCs), a team led by Mount Sinai researchers has gained new insight into genetic changes that may turn a well known anti-cancer signaling gene into a driver of risk for bone cancers, where the survival rate has not improved in 40 years despite treatment advances.
The study results, published today in the journal Cell, revolve around iPSCs, which since their 2006 discovery have enabled researchers to coax mature (fully differentiated) bodily cells (e.g. skin cells) to become like embryonic stem cells. Such cells are pluripotent, able to become many cell types as they multiply and differentiate to form tissues. The iPSCs can then be converted again as needed into differentiated cells such as heart muscle, nerve cells, bone, etc.
While some seek to use iPSCs as replacements for cells compromised by disease, the new Mount Sinai study sought to determine if they could serve as an accurate model of genetic disease "in a dish." In this context, the dish stands for a self-renewing, unlimited supply of iPSCs or a cell line - which enables in-depth study of disease versions driven by each person's genetic differences. When matched with patient records, iPSCs and iPSC-derived target cells may be able to predict a patient's prognosis and whether or not a given drug will be effective for him or her.
In the current study, skin cells from patient with and without disease were turned into patient-specific iPSC lines, and then differentiated into bone-making cells where both rare and common bone cancers start. This new bone cancer model does a better job than previously used mouse or cellular models of "recapitulating" the features of bone cancer cells driven by key genetic changes.
"Our study is among the first to use induced pluripotent stem cells as the foundation of a model for cancer," said lead author Dung-Fang Lee, PhD, a postdoctoral fellow in the Department of Developmental and Regenerative Biology, Icahn School of Medicine at Mount Sinai. "This model, when combined with a rare genetic disease, revealed for the first time how a protein known to prevent tumor growth in most cases, p53, may instead drive bone cancer when genetic changes cause too much of it to be made in the wrong place."
Rare Disease Sheds Light on Common Disease
The Mount Sinai disease model research is based on the fact that human genes, the DNA chains that encode instructions for building the body's structures and signals, randomly change all the time. As part of evolution, some code changes, or mutations, make no difference, some confer advantages, and others cause disease. Beyond inherited mutations that contribute to cancer risk, the wrong mix of random, accumulated DNA changes in bodily (somatic) cells as we age also contributes to cancer risk.
The current study focused on the genetic pathways that cause a rare genetic disease called Li-Fraumeni Syndrome or LFS, which comes with high risk for many cancers in affected families. A common LFS cancer type is osteosarcoma (bone cancer), with many diagnosed before the age of 30. Beyond LFS, osteosarcoma is the most common type of bone cancer in all children, and after leukemia, the second leading cause of cancer death for them.
Importantly, about 70 percent of LFS families have a mutation in their version of the gene TP53, which is the blueprint for protein p53, well known by the nickname "the tumor suppressor." Common forms of osteosarcoma, driven by somatic versus inherited mutations, have also been closely linked by past studies to p53 when mutations interfere with its function.
Rare genetic diseases like LFS are good study models because they tend to proceed from a change in a single gene, as opposed to many, overlapping changes seen in more related common diseases, in this case more common, non-inherited bone cancers. The LFS-iPSC based modeling highlights the contribution of p53 alone to osteosarcoma.
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