Category Archives: DNA

2000 miles apart
READ STORYLINE BEFORE YOU WATCH: A world changing secret organization called the OPP (Over population project); makes everyone takes serum and pills before they go to bed that night, so EVERYONE in the world does. What happens when they wake up, everyone on the earth is separated 2000 miles apart ( some are even unlucky and are placed dna in the ocean) BUT 2 girls (Hannah and Ali) have glitches and only are 2 miles apart. They try yelling at each other and finding each other, but Ali dies of starvation right before Hannah can find her. Ok so this is really confusing, but everyone seems to like my storylines the best so yay. 🙂 -DrewFrom:drewberryxViews:23 7ratingsTime:01:08More inMusic

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2000 miles apart - Video

Future - DNA [Prod. By 808 Mafia]
Future - DNA [Prod. By 808 Mafia] *NEW*From:scottdonta20Views:0 0ratingsTime:03:12More inMusic

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Future - DNA [Prod. By 808 Mafia] - Video

Little Mix drop in to the Official Chart
Little Mix join Reggie on the Official Chart to find out where their latest single, DNA, has made it. Catch the Official Chart every Sunday on Radio 1. Listen from 4, watch from 6 http://www.bbc.co.ukFrom:bbcradio1Views:148 29ratingsTime:05:00More inEntertainment

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Little Mix drop in to the Official Chart - Video

"DNA" by Little Mix Acoustic Cover
Bobby and I put this together in one hour.From:denikkixheartsxyouViews:2 0ratingsTime:03:46More inMusic

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"DNA" by Little Mix Acoustic Cover - Video

The tiny, all-female bdelloid rotifers have endured the past 80 million years without sex. New research shows that gobbling up foreign DNA from other simple life-forms might be the asexual animal's secret to survival.

In the study, scientists discovered that up to 10 percent of the active genes in microscopic bdelloids comes from bacteria and other organisms like fungi and algae. The finding adds to "the weirdness of an already odd little creature," said Alan Tunnacliffe, a University of Cambridge professor and lead author of the study.

"We don't know how the gene transfer occurs, but it almost certainly involves ingesting DNA in organic debris, which their environments are full of," Tunnacliffe explained in a statement. "Bdelloids will eat anything smaller than their heads!"

Many asexual creatures are thought to be doomed to extinction due to the lack of genetic diversity and build-up of mutations that often come with reproducing from just one parent's DNA. But bdelloids have managed to avoid such pitfalls of asexual life, diversifying into at least 400 species.

One of the critters' more remarkable qualities is their ability to withstand extreme dehydration, which could be, in part, thanks to the alien DNA. The new study found that some of the foreign genes are activated when the bdelloids begin to dry out in their ephemeral aquatic homes. These genes also might be behind powerful antioxidants thought to protect bdelloids from the by-products of drying out.

"These antioxidants have not yet been identified, but we think that some of them result from foreign genes," Tunnacliffe added.

Bdelloids' success could also be attributed to their potent DNA repair mechanisms, which seem to have evolved thanks to a duplicate set of genes, according to research detailed in 2008 in the journal Proceedings of the National Academy of Sciences.

The new findings were detailed in the journal PLoS Genetics Thursday (Nov. 15).

Follow LiveScience on Twitter @livescience. We're also on Facebook & Google+.

2012 LiveScience.com. All rights reserved.

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'Odd little creature' skips sex, eats DNA

Michael agreed to submit to a DNA test for an episode of "Trisha Goddard" -- where he comes face-to-face for the first time with the 17-year-old girl who claimed to be his daughter -- and the results came back positive.

The girl's mother -- Kristi Horn -- has claimed Michael was the father for years, and has even tried multiple times to get him to pay child support ... but Michael adamantly denied the allegations.

The reveal -- which airs tonight on NBC -- is cringeworthy ... Michael can't believe the news ... and the girl's mother immediately bursts into tears.

After, Michael gets up to hug his newfound daughter (Ashley) and she resists, shouting, "No! No! No!" while Michael pleads, "I just want to hug you."

Michael's two youngest children with Dina -- Ali and Cody -- were born in 1993 and 1996 respectively. Which means Ashley was conceived somewhere in the middle. - more on this story

TMZ submitted this story. Copyright TMZ - Excerpted here with permission.

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DNA Test Proves Michael Lohan Father of Lovechild

ScienceDaily (Nov. 16, 2012) A new discovery from researchers at Huntsman Cancer Institute (HCI) at the University of Utah concerning a fundamental understanding about how DNA works will produce a "180-degree change in focus" for researchers who study how gene packaging regulates gene activity, including genes that cause cancer and other diseases.

The discovery, by Bradley R. Cairns, PhD, Senior Director of Basic Science at HCI and a professor in the Department of Oncological Sciences, is reported in this week's online issue of the journal Nature.

Cairns's research focuses on chromatin remodeling complexes (CRCs), which are cellular protein complexes that behave like motors, expanding or compacting different portions of DNA to either express or silence genes, respectively. Before, scientists thought that the motor within CRCs waits at rest until it receives instructions. Cairns and co-author Cedric R. Clapier show that the motor within a key CRC responsible for gene packaging and assembly is intrinsically turned on, and instead requires specific instructions to turn it off.

"Many articles in the research literature show that CRCs are mutated in cancer cells. They are intimately involved in regulating gene expression -- responsible for correctly packaging genes that control growth proliferation and for unpackaging tumor suppressors," said Cairns. "This research reveals principles by which CRC mutations could cause cancer."

Chromosomes are made of long DNA strands compressed around nodes of protein called nucleosomes; when DNA is compressed, the genes in that area are turned off. Some CRCs, called disassembly CRCs, act as motors that unwind sections of DNA chains, making genes active for a given cell process. Another type, called assembly CRCs, rewinds the DNA chain, recompressing it when the process is complete. The unwind-rewind cycle is repeated continuously throughout a cell's life.

In this study, Cairns and Clapier focused on assembly CRCs. "Before this research, we thought that the motor was off unless a protein coming from another part of the cell turned it on," said Cairns. "Researchers have been searching for the switch by looking at the CRC motor to see what binds to it.

"As it turns out, we discovered that the CRC motor already carries on its flank a 'switch' that inhibits its action until a marker sequence, located on the nucleosome, is encountered. The marker flips the inhibitor switch and allows the CRC to crank the DNA chain back around the nucleosome, promoting gene packaging and silencing" Cairns said. "Our results change where future researchers should be looking to understand how CRCs are regulated -- not at the CRC motor itself, but at the 'switches' that flank the motor."

The study also describes a measuring function on the CRC that checks for the correct distance between one nucleosome and the next, telling the motor to switch off at the proper time, a function needed for gene silencing.

Cairns's lab will now examine this same switching concept in disassembly remodelers. "There are additional remodeler families with alternative functions, like DNA repair," said Cairns. "We think this concept will apply to them as well."

This research was supported by funding from the National Institutes of Health (GM60415 and CA042014) and from the Howard Hughes Medical Institute.

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DNA packaging discovery reveals principles by which CRC mutations may cause cancer

The tiny, all-female bdelloid rotifers have endured the past 80 million years without sex. New research shows that gobbling up foreign DNA from other simple life-forms might be the asexual animal's secret to survival.

In the study, scientists discovered that up to 10 percent of the active genes in microscopic bdelloids comes from bacteria and other organisms like fungi and algae. The finding adds to "the weirdness of an already odd little creature," said Alan Tunnacliffe, a University of Cambridge professor and lead author of the study.

"We don't know how the gene transfer occurs, but it almost certainly involves ingesting DNA in organic debris, which their environments are full of," Tunnacliffe explained in a statement. "Bdelloids will eat anything smaller than their heads!"

Many asexual creatures are thought to be doomed to extinction due to the lack of genetic diversity and build-up of mutations that often come with reproducing from just one parent's DNA. But bdelloids have managed to avoid such pitfalls of asexual life, diversifying into at least 400 species.

One of the critters' more remarkable qualities is their ability to withstand extreme dehydration, which could be, in part, thanks to the alien DNA. The new study found that some of the foreign genes are activated when the bdelloids begin to dry out in their ephemeral aquatic homes. These genes also might be behind powerful antioxidants thought to protect bdelloids from the by-products of drying out.

"These antioxidants have not yet been identified, but we think that some of them result from foreign genes," Tunnacliffe added.

Bdelloids' success could also be attributed to their potent DNA repair mechanisms, which seem to have evolved thanks to a duplicate set of genes, according to research detailed in 2008 in the journal Proceedings of the National Academy of Sciences.

The new findings were detailed in the journal PLoS Genetics Thursday (Nov. 15).

Follow LiveScience on Twitter @livescience. We're also on Facebook& Google+.

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'Odd Little Creature' Skips Sex and Eats DNA

Public release date: 18-Nov-2012 [ | E-mail | Share ]

Contact: Karen N. Peart karen.peart@yale.edu 203-432-1326 Yale University

The prevailing wisdom has been that every cell in the body contains identical DNA. However, a new study of stem cells derived from the skin has found that genetic variations are widespread in the body's tissues, a finding with profound implications for genetic screening, according to Yale School of Medicine researchers.

Published in the Nov. 18 issue of Nature, the study paves the way for assessing the extent of gene variation, and for better understanding human development and disease.

"We found that humans are made up of a mosaic of cells with different genomes," said lead author Flora Vaccarino, M.D., the Harris Professor of Child Psychiatry at the Yale Child Study Center. "We saw that 30 percent of skin cells harbor copy number variations (CNV), which are segments of DNA that are deleted or duplicated. Previously it was assumed that these variations only occurred in cases of disease, such as cancer. The mosaic that we've seen in the skin could also be found in the blood, in the brain, and in other parts of the human body."

The longstanding belief has been that our cells have the same DNA sequence and this blueprint governs the body's functions. The Yale team's research challenges this dogma. Some scientists have hypothesized that during development, when DNA is copied from mother to daughter cells, there could be deletions, duplications and changes in the sequence of the DNA, and an entire group of genes could be affected. This premise has been incredibly difficult to test, but Vaccarino and colleagues have done so in this new study.

The team used whole genome sequencing to study induced pluripotent stem cells lines (iPS), which are stem cells developed from a mature-differentiated cell. The team grew cells taken from the inner upper arms of two families. The team spent two years characterizing these iPS cell lines and comparing them to the original skin cells.

While observing that the genome of iPS cells closely resembles the genome of skin cells from which they originated, the team could identify several deletions or duplications involving thousands of base pairs of DNA. The team then performed additional experiments to understand the origin of those differences, and showed that at least half of them pre-existed in small fractions of skin cells. These differences were revealed in iPS cells because each iPS line is derived from one, or very few, skin cells. Vaccarino said these iPS lines could act as a magnifying glass to see the mosaic of genomic differences in the body's cells.

"In the skin, this mosaicism is extensive and at least 30 percent of skin cells harbor different deletion or duplication of DNA, each found in a small percentage of cells," said Vaccarino. "The observation of somatic mosaicism has far-reaching consequences for genetic analyses, which currently use only blood samples. When we look at the blood DNA, it's not exactly reflecting the DNA of other tissues such as the brain. There could be mutations that we're missing."

"These findings are shaping our future studies, and we're doing more studies of the developing brains of animals and humans to see if this variation exists there as well," Vaccarino added.

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Skin cells reveal DNA's genetic mosaic

Posted on November 18, 2012, Sunday

Guanine, adenine, thymine, cytosine Android? The Droid DNA the latest addition to Verizons Droid series may not contain any actual nucleotides (that we know of), but that doesnt make this HTC-made superphone any less of a powerhouse. On the contrary, weve been eyeballing this handset with eager anticipation ever since it first launched in Japan as theJ Butterfly; much like its counterpart from the Land of the Rising Sun, the DNA boasts a jaw-dropping 5-inch,1080p display. But while that may be the headliner-worthy feature, you certainly cant go wrong with a quad-core Snapdragon S4 Pro processor and 2GB of RAM taking charge behind the scenes, along with anImageSensecamera and other top-notch specs.

In certain respects, the Droid DNA is a sneak preview of whats to come in 2013: a wave of high-performance superphones that take advantage of this improved resolution, and offer a long list of other top-notch features. Indeed, thats a future we could all definitely live with, but lets not get too ahead of ourselves; weve got a phone to review, after all. Is the display as tantalizing as it sounds? Will its size be a selling point or a major distraction? Should you waltz into a Verizon store on Black Friday and demand they take your hard-earned $200? Follow us after the break as we focus on the here and now.

HTC may not be the king of the hill in device sales or financials, but 2012 has convinced us that its firmly planted as the champion in smartphone design. The companys had a knack for thinking outside the rectangular box and coming up with cleverly built handsets theSensationseries andTouch Diamondcome to mind but it seems to have cranked the focus up another notch this year. The One series was beautifully crafted and each model therein acted as a brilliant indication of HTCs renewed dedication; without skipping a beat, it was later followed up with the colorful and visually stunningWindows Phone 8X, which has been readily endorsed by Microsoft.

This brief bit of background was added to emphasize one important thing: HTC isnt slacking off with the Droid DNA. Everything about the phone straight down to the Ferrari-inspired edges screams luxury and ensures a comfortable experience. Were just as drawn to its beauty as we were when we first beheld theOne Xwith our own eyes. Its an incredibly sleek and solidly built device that wont leave us worried about durability, since it consists of aluminum on the sides and a soft-touch polycarbonate on the back that is quite similar to the material used on the 8X. (Unfortunately, its also a fingerprint magnet.)Gorilla Glass 2graces the display with the promise of preventing that beautiful screen from getting scuffed.

Currently, the DNA only comes in one color option: black with red accents. These two colors have been HTCs bread and butter for many years, so it comes as no huge shocker that this would be the default choice for, well, Big Red. It also correlates with theBeats Audiocolor scheme, so its a perfect combination for every party involved. The red isnt overbearing and complements the black quite well, accentuating the power button, earpiece, camera and edges (as we mentioned earlier, the grille-like edge design was apparently inspired by Ferrari, and we fully support that decision).

The same folks who were concerned about theGalaxy Note IIsbehemoth size will also have identical reservations for the 5-inch DNA at first, but this particular device isnt looking to compete in the same genre. HTC went out of its way to emphasize that the DNA is a smartphone, not a phablet, and we can definitely attest to that statement. Holding it up to your ear when making a call likely wont inspire the same feelings of awkwardness as the Note II might do, and its much easier to use for one-handed tasks without needing special keyboards or dialpads. In fact, the DNAs form factor reminds us of what youll find on the One X andSamsung Galaxy S III: at 70.5mm (2.78 inches) wide, its only 0.6mm wider than the One X and is actually 0.1mmnarrowerthan the GS3. Boasting a height of 141mm (5.55 inches), its also taller than either phone. At its thickest point, the DNA measures at 9.73mm (0.38 inch), which is a bit thicker than the 8.9mm One X and 8.6mm GS3, but the back gently slopes to meet the 4mm-thick tapered edges. We were expecting the phone to require some sort of adjustment period at first, but we found the DNA to be a completely natural fit in the palm of our hand and it feels great.

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HTC Droid DNA review